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Thus, TB-500 may be hypothesized to support angiogenesis by combining a cytoskeleton-linked increase in endothelial motility with signaling changes that raise pro-angiogenic programs (VEGF-A and Ang2/Tie2) via Notch/NF-B coupling in damaged cellular structure

[] While extreme heat exposure results in acute kidney injury, a strong case for the existence of heat stress nephropathy and its role in the causation of CKDu has been the topic of interest in various studies, as detailed in Table 1

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Reference lists of included articles and relevant reviews were manually screened to identify additional pertinent studies